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Acute coronary syndrome, fun, fast, easy!!!

Acute coronary syndrome, fun, fast, easy!!!

November 2, 2016 @ 11:07 am
by Heart News 247

Acute Coronary Syndrome, Fun, Fast, Easy!!!

Acute coronary syndrome is a term used by any condition brought on by sudden reduced blood flow to the heart. You have some sort of chest pain that is ischemic in nature but you don’t know why. segment elevation myocardial infarction. A major artery in the heart is completely blocked and when its completely blocked . When an artery is completely blocked there is lack no blood flow to that area and the muscle it generally affects large areas of heart muscle causing st segment elevation. These are the patients that need door to balloon time with 90 minutes or less. What does this mean? PCI is the is always preferred over fibrinolytics.
PCI is a percutaneous coronary intervention in which a catheter with a deflated balloon is inserted into the artery usually femoral or radial artery until it reaches the site of blockage. Xray imaging is used to guied the wire. When the catheter reaches the blocked areas a balloon is inflated to open the artery a stent is often placed to permanently keep the artery open.

Acute Coronary Syndrome, Fun, Fast, Easy!!!

Nancy the NSTEMI arteries are not usually completely blocked but there is heart muscle damage and you know this not by EKG but cardiac enzymes!! Serial cardiac enzymes is the key here. They could be normal initially but could rise in the 2nd or 3rd set.

Acute Coronary Syndrome, Fun, Fast, Easy!!!

Ursela- the unstable angina is no st elevation, normal cardiac enzymes. but they’ll complain of an onset of pain that will make you think it could be their heart. Ex, new onset of chest pain, pain while at rest, or minimal exertion. The worst thing about unstable angina is that it could conver to stemi or nstemi.

Acute Coronary Syndrome, Fun, Fast, Easy!!!

Signs and symptoms
Chest pain, dizziness, lightheadness, shortness of breath, chest pain at rest, worsening chest pain, chest pain with minimal exretion

Acute Coronary Syndrome, Fun, Fast, Easy!!!

Diagnosis
EKG ST ELEVATION- SAM STEMI
Cardiac enzymes + Nancy NSTEMI
– Enzymes – Ursela unstable angina

Acute Coronary Syndrome, Fun, Fast, Easy!!!

Treatment
ASA, BB lowers mortality
NSTEMI/USA-antcoagulant therapy- help prevent that artery from completely clotting off. No thrombolytics
Heparin- prevents clots from forming,
GlycoprotieN –
Nitrates
Plavix
Angioplasty within 72 hours of admission

Acute Coronary Syndrome, Fun, Fast, Easy!!!

STEMI- tpa, emergent cath
Nitrates – relieve and prevent ischemia by improving blood flow
Urgent angioplasty BETA BLOCKER ARE FOR EVERYONE BUT THROMBOLYTICS ARE NOT

Acute Coronary Syndrome, Fun, Fast, Easy!!!

myocardial tissue.
In most cases ischemic heart disease seems to progress through stages of from fatty streak deposits in coronary arteries to development of plaque, which increases in size until it causes luminal obstruction
It’s a serious form of a heart attack when the coronary arteries are blocked resulting in death in the region supplied by that artery. Depending on where the heart attack occurs the symptoms can be minimal to death. About 1.2 million people a year experience a heart attack and every minute one American will die of coronary artery disease.
A STEMI is different from a NSTEMI in there is no ST segment elevation
The difference between STEMI and NSTEMI is based on EKG findings. A person is still having a heart attack in both cases but STEMI typically implies there is bigger heart muscle at risk , more occlusion compared to NSTEMI.
STEMIs require emergent catherization whereas NSTEMIs may or may not depending on severity of symptoms, ekg findings etc.

NSTEMI arteries are not usually completely blocked but there is heart muscle damage and you know this not by EKG but cardiac enzymes!! Serial cardiac enzymes is the key here. They could be normal initially but could rise in the 2nd or 3rd set.

PCI is a percutaneous coronary intervention in which a catheter with a deflated balloon is inserted into the artery usually femoral the blocked areas a balloon is inflated to open the artery a stent is often placed to permanently keep the artery open.
The patency with PCI is 90% or higher whereas the patency with fibrinolytics is 65% and recurrent reocclusion is more common
The goal of reperfusion is a door of ED to balloon inflation time of less than 90 minutes.
Fibrolytic theray can be given to break up clots in patients within 30 mins if PCI is unavailable but remember remember PCI is better.

Acute coronary syndrome, fun, fast, easy!!!

Acute coronary syndrome is a term used by any condition brought on by sudden reduced blood flow to the heart. You have some sort of chest pain that is ischemic in nature but you don’t know why. segment elevation myocardial infarction. A major artery in the heart is completely blocked and when its completely blocked . When an artery is completely blocked there is lack no blood flow to that area and the muscle it generally affects large areas of heart muscle causing st segment elevation. These are the patients that need door to balloon time with 90 minutes or less. What does this mean? PCI is the is always preferred over fibrinolytics.
PCI is a percutaneous coronary intervention in which a catheter with a deflated balloon is inserted into the artery usually femoral or radial artery until it reaches the site of blockage. Xray imaging is used to guied the wire. When the catheter reaches the blocked areas a balloon is inflated to open the artery a stent is often placed to permanently keep the artery open.

Nancy the NSTEMI arteries are not usually completely blocked but there is heart muscle damage and you know this not by EKG but cardiac enzymes!! Serial cardiac enzymes is the key here. They could be normal initially but could rise in the 2nd or 3rd set.

Ursela- the unstable angina is no st elevation, normal cardiac enzymes. but they’ll complain of an onset of pain that will make you think it could be their heart. Ex, new onset of chest pain, pain while at rest, or minimal exertion. The worst thing about unstable angina is that it could conver to stemi or nstemi.



Signs and symptoms
Chest pain, dizziness, lightheadness, shortness of breath, chest pain at rest, worsening chest pain, chest pain with minimal exretion

Diagnosis
EKG ST ELEVATION- SAM STEMI
Cardiac enzymes + Nancy NSTEMI
- Enzymes – Ursela unstable angina

Treatment
ASA, BB lowers mortality
NSTEMI/USA-antcoagulant therapy- help prevent that artery from completely clotting off. No thrombolytics
Heparin- prevents clots from forming,
GlycoprotieN -
Nitrates
Plavix
Angioplasty within 72 hours of admission

STEMI- tpa, emergent cath
Nitrates – relieve and prevent ischemia by improving blood flow
Urgent angioplasty BETA BLOCKER ARE FOR EVERYONE BUT THROMBOLYTICS ARE NOT

myocardial tissue.
In most cases ischemic heart disease seems to progress through stages of from fatty streak deposits in coronary arteries to development of plaque, which increases in size until it causes luminal obstruction
It’s a serious form of a heart attack when the coronary arteries are blocked resulting in death in the region supplied by that artery. Depending on where the heart attack occurs the symptoms can be minimal to death. About 1.2 million people a year experience a heart attack and every minute one American will die of coronary artery disease.
A STEMI is different from a NSTEMI in there is no ST segment elevation
The difference between STEMI and NSTEMI is based on EKG findings. A person is still having a heart attack in both cases but STEMI typically implies there is bigger heart muscle at risk , more occlusion compared to NSTEMI.
STEMIs require emergent catherization whereas NSTEMIs may or may not depending on severity of symptoms, ekg findings etc.

NSTEMI arteries are not usually completely blocked but there is heart muscle damage and you know this not by EKG but cardiac enzymes!! Serial cardiac enzymes is the key here. They could be normal initially but could rise in the 2nd or 3rd set.


PCI is a percutaneous coronary intervention in which a catheter with a deflated balloon is inserted into the artery usually femoral the blocked areas a balloon is inflated to open the artery a stent is often placed to permanently keep the artery open.
The patency with PCI is 90% or higher whereas the patency with fibrinolytics is 65% and recurrent reocclusion is more common
The goal of reperfusion is a door of ED to balloon inflation time of less than 90 minutes.
Fibrolytic theray can be given to break up clots in patients within 30 mins if PCI is unavailable but remember remember PCI is better.

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Comments

  • Jackie Lou Pelayo
    May 21, 2016

    Thanks! Concise but very helpful😊

  • Ayo Renner
    June 7, 2016

    GOD BLESS YOUR GOOD HAIR

  • Sunnychuu
    June 13, 2016

    lol I love the way you talk

  • st kings ox
    July 25, 2016

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  • Alison Loughery
    August 27, 2016

    I love this! I actually learned something and had a giggle at the same time
    😄

  • Matthew Curran
    September 22, 2016

    I thought her voice was very annoying for the first minute then I started
    cracking up

  • Glory
    October 6, 2016

    Love this! Thanks

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